SCIENTIFIC EVIDENCE

Stress-Programmed Immune Niches Fuel TNFR2+ Treg Activation and Drive Neoadjuvant Chemotherapy Resistance in Breast Cancer.

Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Shao Z, Wang X, Zhu H, Yao L, Zhang Q, Zhai Z, Lv X, Xia X, Zhang Y, Lu X, Quan X, Yang J, Li X, Dong J, Shao ZM, Wang R, Xiu B, Wu J, Chen S.

Publication Overview

Key findings, Countstar context, and access to the original paper.
2026
Cancer Research
Immunology
Single-Cell Analysis
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Key Finding
The findings highlight the immune-editing role of stress-programmed Effector regulatory T cells, Neoadjuvant chemotherapy, Stress-programmed immune states, TNF α -TNFR2 axis, breast cancercell states and support their therapeutic potential as a rational target in breast cancer.
Countstar Connection
The researchers assessed cell numbers in the study's cell samples with Countstar Rigel S2 during preparation for single-cell or single-nucleus analysis.
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