SCIENTIFIC EVIDENCE

A-MYB substitutes for B-MYB in activating cell cycle genes and in stimulating proliferation.

Nucleic acids research
Kohler R, Engeland K.

Publication Overview

Key findings, Countstar context, and access to the original paper.
2024
Cell Counting & Viability
No items found.
Key Finding
Knockdown experiments in cells not related to spermatogenesis show that B-MYB loss alone merely delays cell cycle progression. In summary, the authors demonstrate that A-MYB is an activator of the mitotic cell cycle by activating late cell cycle genes.
Countstar Connection
Countstar (model not specified) provided cell-count measurements for the study's cell samples during the study's growth assessment.
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